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Adjunct Faculty

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Bernstein, Kara
Associate Professor
Repair of DNA damage is crucial to prevent accumulation of mutations that can cause human disease, such as cancer. Our lab studies how double-strand breaks in the DNA, one of the most lethal types of DNA lesions, are repaired. By understanding the ...
Bonacci, Gustavo
Adjunct Assistant Professor
Carroll, Bryan
Adjunct Associate Professor
Cavanaugh, Jane
Adjunct Research Assistant Professor
Conrads, Thomas
Adjunct Associate Professor
Du, Wei
Adjunct Associate Professor
Dubey, Raghvendra
Adjunct Professor
Edreira, Martin
Adjunct Research Instructor
Flint, Melanie
Adjunct Research Assistant Professor
Fogle, Keri
Research Instructor
Molecular mechanisms of metabolic modulation of neuronal excitability, pharmacological and dietary interventions for neurological consequences of metabolic disease
Hrizo, Stacy
Adjunct Assistant Professor
Hu, Baoli
Assistant ProfessorDirector, Brain Tumor Evolution & Therapy Lab
Karim, Md Fazlul
Postdoctoral Associate
Kurnikova, Maria
Adjunct Associate Professor
Liu, Yongjian
Adjunct Associate Professor
Nichols, Mark
Adjunct Assistant Professor
Puthenveedu, Manojkumar
Adjunct Assistant Professor
Radi, Rafael
Adjunct Professor
Rizzo, Stacey
Associate Professor of NeurobiologyDirector, Preclinical Phenotyping Core
Romesberg, Amy
Adjunct Assistant Professor
Rubbo, Homero
Adjunct Professor
Rudolph, Tanja
Adjunct Assistant Professor
Rudolph, Volker
Adjunct Assistant Professor
Siegfried, Jill
Adjunct Professor
Sobol, Robert
Adjunct Professor
Research focuses on the role of DNA repair proteins in the repair of DNA damage induced by chemotherapeutic drugs and radiation, with an emphasis on the cell signaling and the cell survival pathways that are engaged by such DNA damage.
Sutkeviciute, Ieva
Research Assistant Professor
Tarpey, Margaret
Adjunct Professor
Trostchansky, Andres
Adjunct Associate Professor
Tucker, Kristal
Research Instructor
Wang, Bin
Adjunct Assistant Professor
Yalowich, Jack
Adjunct Professor
Zhang, Lin
Professor
Genetic basis of differential responses to anticancer drugs and mechanisms of drug-induced apoptosis.